1. Immunological analysis of a patient with hepatitis B virus (HBV) reactivation after bone marrow transplantation.

    Internal Medicine (Tokyo, 1992) 54(10):1213 (2015) PMID 25986258

    Patients with resolved hepatitis B virus (HBV) infection undergoing chemo- or immunosuppressive therapy are at potential risk for HBV reactivation. To determine whether the host immune response contributes to liver injury, we performed an immunological analysis of a patient with HBV reactivation...
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  2. Blockade of indoleamine 2,3-dioxygenase reduces mortality from peritonitis and sepsis in mice by regulating functions of CD11b+ peritoneal cells.

    Infection and Immunity 82(11):4487 (2014) PMID 25114116

    Indoleamine 2,3-dioxygenase-1 (Ido), which catalyzes the first and limiting step of tryptophan catabolism, has been implicated in immune tolerance. However, the roles of Ido in systemic bacterial infection are complicated and remain controversial. To explore this issue, we examined the roles of ...
  3. Blockade of indoleamine 2,3-dioxygenase reduces mortality from peritonitis and sepsis in mice by regulating functions of CD11b+ peritoneal cells.

    Infection and Immunity 82(11):4487 (2014) PMID 25114116

    Indoleamine 2,3-dioxygenase-1 (Ido), which catalyzes the first and limiting step of tryptophan catabolism, has been implicated in immune tolerance. However, the roles of Ido in systemic bacterial infection are complicated and remain controversial. To explore this issue, we examined the roles of ...
  4. Blockade of indoleamine 2,3-dioxygenase reduces mortality from peritonitis and sepsis in mice by regulating functions of CD11b+ peritoneal cells.

    Infection and Immunity 82(11):4487 (2014) PMID 25114116 PMCID PMC4249307

    Indoleamine 2,3-dioxygenase-1 (Ido), which catalyzes the first and limiting step of tryptophan catabolism, has been implicated in immune tolerance. However, the roles of Ido in systemic bacterial infection are complicated and remain controversial. To explore this issue, we examined the roles of ...
  5. Blockade of indoleamine 2,3-dioxygenase reduces mortality from peritonitis and sepsis in mice by regulating functions of CD11b+ peritoneal cells.

    Infection and Immunity 82(11):4487 (2014) PMID 25114116 PMCID PMC4249307

    Indoleamine 2,3-dioxygenase-1 (Ido), which catalyzes the first and limiting step of tryptophan catabolism, has been implicated in immune tolerance. However, the roles of Ido in systemic bacterial infection are complicated and remain controversial. To explore this issue, we examined the roles of ...
  6. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014)

    Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties.
  7. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014)

  8. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014)

  9. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014) PMID 24642363

    Graphical abstract
  10. Synthesis of hydroxycinnamoyl β-D-xylopyranosides and evaluation of their antioxidant properties.

    Carbohydrate Research 388:138 (2014) PMID 24642363

    Various hydroxycinnamoyl β-d-xylopyranosides were efficiently prepared from 2,3,4-tri-O-acetyl-α-d-xylopyranosyl bromide (TAXB) with amine by amine-promoted glycosylation. The resulted acetylated hydroxycinnamoyl β-d-xylopyranosides with acetoxy groups at C-2, C-3, and C-4 were regioselectively ...
  11. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014)

  12. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014)

  13. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014)

    Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties.
  14. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014)

  15. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014)

    Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties.
  16. Synthesis of hydroxycinnamoyl β-d-xylopyranosides and evaluation of their antioxidant properties

    Carbohydrate Research 388:138 (2014)

  17. Liver acid sphingomyelinase inhibits growth of metastatic colon cancer.

    Journal of Clinical Investigation 123(2):834 (2013) PMID 23298833 PMCID PMC3561803

    Acid sphingomyelinase (ASM) regulates the homeostasis of sphingolipids, including ceramides and sphingosine-1-phosphate (S1P). These sphingolipids regulate carcinogenesis and proliferation, survival, and apoptosis of cancer cells. However, the role of ASM in host defense against liver metastasis...
  18. Liver acid sphingomyelinase inhibits growth of metastatic colon cancer.

    Journal of Clinical Investigation 123(2):834 (2013) PMID 23298833 PMCID PMC3561803

    Acid sphingomyelinase (ASM) regulates the homeostasis of sphingolipids, including ceramides and sphingosine-1-phosphate (S1P). These sphingolipids regulate carcinogenesis and proliferation, survival, and apoptosis of cancer cells. However, the role of ASM in host defense against liver metastasis...
  19. A case of malignant melanoma simultaneously found in the lung and small intestine.

    Clinical Journal of Gastroenterology 6(1):25 (2013) PMID 26181400

    Metastatic melanoma in the small bowel is a common cause of secondary intestinal tumors. We present a case of a 77-year-old man with melena resulting from melanoma in the small bowel that was simultaneously found with malignant melanoma in the lung. Abdominal contrast-enhanced computed tomograph...
  20. Tumor necrosis factor-α promotes cholestasis-induced liver fibrosis in the mouse through tissue inhibitor of metalloproteinase-1 production in hepatic stellate cells.

    PLoS ONE 8(6):e65251 (2013) PMID 23755201 PMCID PMC3670853

    Tumor necrosis factor (TNF)-α, which is a mediator of hepatotoxicity, has been implicated in liver fibrosis. However, the roles of TNF-α on hepatic stellate cell (HSC) activation and liver fibrosis are complicated and remain controversial. To explore this issue, the role of TNF-α in cholestasis-...