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Journal Of Cellular Physiology

Print ISSN
0021-9541
Electronic ISSN
1097-4652
Impact factor
3.986
Publisher
wiley
URL
http://onlinelibrary.wiley.com/journal/10.1002/(ISSN)1097-4652
Usage rank
107
Article count
11556
Free count
400
Free percentage
0.0346141
PDFs via platforms
Wiley

  1. P53 dependent mitochondrial permeability transition pore opening is required for dexamethasone-induced death of osteoblasts.

    Journal Of Cellular Physiology 229(10):1475 (2014) PMID 24615518

    We studied the cellular mechanism of dexamethasone (Dex)-induce osteoblast cell death by focusing on the role of mitochondrial permeability transition pore (mPTP). In cultured osteoblastic MC3T3-E1 cells, Dex-induced mPTP opening, which was demonstrated by mitochondrial membrane potential (MPP) decr...
  2. Calcineurin Regulates Progressive Motility Activation of Rhinella (Bufo) arenarum Sperm Through Dephosphorylation of PKC Substrates.

    Journal Of Cellular Physiology 229(10):1378 (2014) PMID 24648036

    We have shown that the signal transduction pathway required for in situ motility activation involves a rise in intracellular cAMP through a transmembrane adenylyl cyclase and activation of PKA, mostly in the midpiece and in the sperm head. In this report, we demonstrate that activation of calcineuri...
  3. Quantifying mitotic chromosome dynamics and positioning.

    Journal Of Cellular Physiology 229(10):1301 (2014) PMID 24683081

    We will discuss analytical approaches to investigate the position-dependent control of mitotic chromosomes in cultured cells. These methods can be used to dissect the specific contributions of mitotic proteins to the molecular control of chromosome dynamics. J. Cell. Physiol. 229: 1301-1305, 2014. ©...
  4. Journal of cellular physiology: volume 229, number 8, august 2014.
    Author(s) unavailable

    Journal Of Cellular Physiology 229(8):C1 (2014) PMID 24756582

    Cover: Diagram showing components of platelet biology important in cancer progression. See article by Sharma et al. on pages 1005-1015. © 2014 Wiley Periodicals, Inc.
  5. Table of contents: volume 229, number 6.
    Author(s) unavailable

    Journal Of Cellular Physiology 229(6):fmi (2014) PMID 24590967

  6. Table of contents: volume 229, number 5.
    Author(s) unavailable

    Journal Of Cellular Physiology 229(5):fmi (2014) PMID 24446202

  7. VEGF inhibitor (Iressa) arrests histone deacetylase expression: single-cell cotransfection imaging cytometry for multi-target-multi-drug ana...

    Journal Of Cellular Physiology 226(8):2115 (2011) PMID 21520063

    We report single-cell cotransfection imaging cytometry to quantitatively screen drug-induced off-target effects. Vascular endothelial growth factor (VEGF) and histone deacetylase (HDAC) genes amplified from the genomic DNA were cloned in fluorescently tagged gene constructs (RFP-HDAC/YFP-VEGF). Thes...
  8. Expression of Notch receptors, ligands, and target genes during development of the mouse mammary gland

    Journal Of Cellular Physiology 226(7):1940 (2011) PMID 21136493

    We have quantitatively determined the levels and mRNA expression patterns of the Notch receptor genes, their ligands and target genes in the postnatal mouse mammary gland. The steady state levels of Notch3 mRNA are the highest among receptor genes, Jagged1 and Dll3 mRNA levels are the highest among...
  9. Numerous isoforms of Fgf8 reflect its multiple roles in the developing brain.

    Journal Of Cellular Physiology 226(7):1722 (2011) PMID 21506104

    We review the roles of Fgf8 in vertebrate development and summarize the recent findings on the in vivo function of different Fgf8 splice variants. We propose that multiple Fgf8 isoform proteins act in concert to regulate the overall function of Fgf8 and account for the diverse and essential role of...
  10. Decreased secretion of MMP by non-lesional late-stage scleroderma fibroblasts after selection via activation of the apoptotic Fas-pathway.

    Journal Of Cellular Physiology 226(7):1907 (2011) PMID 21506121

    Our results show that resistance to apoptosis is an important characteristic of the late-stage lesional SSc fibroblast phenotype. We thus hypothesized that a selection of specific fibroblast subpopulations from late-stage non-lesional SSc skin areas could be at the origin of lesional populations. Th...