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Journal Of Cellular Physiology

Print ISSN
0021-9541
Electronic ISSN
1097-4652
Impact factor
3.986
Publisher
wiley
URL
http://onlinelibrary.wiley.com/journal/10.1002/(ISSN)1097-4652
Usage rank
107
Article count
11556
Free count
400
Free percentage
0.0346141
PDFs via platforms
Wiley

  1. Ribosome biogenesis: emerging evidence for a central role in the regulation of skeletal muscle mass.

    Journal Of Cellular Physiology 229(11):1584 (2014) PMID 24604615

    The ribosome is a supramolecular ribonucleoprotein complex that functions at the heart of the translation machinery to convert mRNA into protein. Ribosome biogenesis is the primary determinant of translational capacity of the cell and accordingly has an essential role in the control of cell growth i...
  2. Thymosin Beta-4 Knockdown in IEC-6 Normal Intestinal Epithelial Cells Induces DNA Re-replication Via Downregulating Emi1.

    Journal Of Cellular Physiology 229(11):1639 (2014) PMID 24615569

    We assessed whether Tβ4 alteration influences normal intestinal epithelial cells because Tβ4 is deemed a novel target for treating colorectal cancer (CRC). For this purpose, we examined the consequences of shRNA-mediated knockdown of Tβ4 in IEC-6 normal rat small intestinal cells and found that inhi...
  3. Biological effects of insulin and its analogs on cancer cells with different insulin family receptor expression.

    Journal Of Cellular Physiology 229(11):1817 (2014) PMID 24683100

    We measured under identical experimental conditions cell proliferation, invasiveness, and foci formation in six cancer cell lines with different insulin receptor family expression levels. The cancer cells studied have a different expression of insulin receptor (IR), its isoforms (IR-A and IR-B), and...
  4. Journal of cellular physiology: volume 229, number 8, august 2014.
    Author(s) unavailable

    Journal Of Cellular Physiology 229(8):C1 (2014) PMID 24756582

    Cover: Diagram showing components of platelet biology important in cancer progression. See article by Sharma et al. on pages 1005-1015. © 2014 Wiley Periodicals, Inc.
  5. Table of contents: volume 229, number 6.
    Author(s) unavailable

    Journal Of Cellular Physiology 229(6):fmi (2014) PMID 24590967

  6. Table of contents: volume 229, number 5.
    Author(s) unavailable

    Journal Of Cellular Physiology 229(5):fmi (2014) PMID 24446202

  7. VEGF inhibitor (Iressa) arrests histone deacetylase expression: single-cell cotransfection imaging cytometry for multi-target-multi-drug ana...

    Journal Of Cellular Physiology 226(8):2115 (2011) PMID 21520063

    We report single-cell cotransfection imaging cytometry to quantitatively screen drug-induced off-target effects. Vascular endothelial growth factor (VEGF) and histone deacetylase (HDAC) genes amplified from the genomic DNA were cloned in fluorescently tagged gene constructs (RFP-HDAC/YFP-VEGF). Thes...
  8. Expression of Notch receptors, ligands, and target genes during development of the mouse mammary gland

    Journal Of Cellular Physiology 226(7):1940 (2011) PMID 21136493

    We have quantitatively determined the levels and mRNA expression patterns of the Notch receptor genes, their ligands and target genes in the postnatal mouse mammary gland. The steady state levels of Notch3 mRNA are the highest among receptor genes, Jagged1 and Dll3 mRNA levels are the highest among...
  9. Numerous isoforms of Fgf8 reflect its multiple roles in the developing brain.

    Journal Of Cellular Physiology 226(7):1722 (2011) PMID 21506104

    We review the roles of Fgf8 in vertebrate development and summarize the recent findings on the in vivo function of different Fgf8 splice variants. We propose that multiple Fgf8 isoform proteins act in concert to regulate the overall function of Fgf8 and account for the diverse and essential role of...
  10. Decreased secretion of MMP by non-lesional late-stage scleroderma fibroblasts after selection via activation of the apoptotic Fas-pathway.

    Journal Of Cellular Physiology 226(7):1907 (2011) PMID 21506121

    Our results show that resistance to apoptosis is an important characteristic of the late-stage lesional SSc fibroblast phenotype. We thus hypothesized that a selection of specific fibroblast subpopulations from late-stage non-lesional SSc skin areas could be at the origin of lesional populations. Th...