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Welcome to the Pubget Medical Subject Headings (MESH) browser. Click on a topic or subtopic below to explore related papers. In the gold box is the most recent paper about the current MESH term, and below are 20 related papers.
Macrophages, Alveolar (0):
Recent article
Macrophages, Alveolar
Abstract
We tested the hypothesis, whether reduced functional receptor availability in mutant mice resulted in increased cellular and molecular inflammatory response during acute inflammation and/or in an impairment of its resolution.
To address this hypothesis we examined the effects of a ca...
|
PMID: 21933390
PDF is available here.
PDF is available here.
| < More recent | Older article > |
Abstract
We tested the hypothesis, whether reduced functional receptor availability in mutant mice resulted in increased cellular and molecular inflammatory response during acute inflammation and/or in an impairment of its resolution.
To address this hypothesis we examined the effects of a ca...
|
PMID: 21933390
PDF is available here.
PDF is available here.
Abstract
We tested the hypothesis, whether reduced functional receptor availability in mutant mice resulted in increased cellular and molecular inflammatory response during acute inflammation and/or in an impairment of its resolution.
To address this hypothesis we examined the effects of a ca...
|
PMID: 21933390
PDF is available here.
PDF is available here.
Abstract
We show that porcine sialoadhesin-specific antibodies and F(ab')₂ fragments trigger sialoadhesin internalization, both in primary porcine macrophages and in cells expressing recombinant porcine sialoadhesin. Using chemical inhibitors, double immunofluorescence stainings and dominant-negative const...
|
PMID: 21359217
PDF is available here.
PDF is available here.
Abstract
We analyzed the steroid sensitivity of ovalbumin (OVA) reactive helper T (Th) cell clones both in vitro and in vivo.
For in vitro experiments, Th clones were cultured with antigen-presenting cells, OVA, and various concentrations of dexamethasone (DEX). The proliferative response of...
|
PMID: 21646805
PDF is available here.
PDF is available here.
Abstract
These results are in agreement with flow cytometry results. ITMC is a valuable tool to monitor the biological responses to nano-sized dosage forms such as NPs. Since the thermal activity of macrophage-NP interactions differed according to the type of NPs used, ITMC may provide a method to better und...
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PMID: 21057907
PDF is available here.
PDF is available here.
Clinical Immunology (137)1 2010
Abstract
We analyzed the M2 marker expression and intracellular signal transduction in the course of cytokine-driven differentiation. We found elevated spontaneous production of the chemokines CCL17, CCL18 and CCL22 and increased expression of CD206 by alveolar macrophages from patients with lung fibrosis. S...
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PMID: 20674506
PDF is available here.
PDF is available here.
Abstract
Lung macrophages, that is, the intravascular, interstitial, pleural, and surface macrophages, are part of the mononuclear phagocyte system. They are derived from the hematopoietic stem cell in the bone marrow with the monocytes as their putative precursors. Macrophages residing on the inner surfaces...
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PMID: 20109124
PDF is available here.
PDF is available here.
Abstract
We tested the postulate that metal import by DMT1 would participate in preventing lung injury after exposure to bleomycin. Microcytic anemia (mk/mk) mice defective in DMT1 and wild-type mice were exposed to either bleomycin or saline via intratracheal instillation and the resultant lung injury was c...
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PMID: 20336479
PDF is available here.
PDF is available here.
Pharmaceutical Research (27)8 2010
Abstract
These results demonstrated the active targeting ability of this kind of mannan-modified DNA-loaded vehicles, which may have great potential for targeted gene delivery....
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PMID: 20422265
PDF is available here.
PDF is available here.
Abstract
We aimed to assess the utility of the lipid index in identifying patients with significant GORD.
Our investigation was a prospective analysis of 34 lung transplant patients who were transplanted between April 1999 and July 2006 at a single institution. All patients with recurrent respiratory infecti...
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PMID: 20466562
PDF is available here.
PDF is available here.
Inflammation Research (59)8 2010
Abstract
Our data demonstrate that phage display technology is a powerful tool for the identification of bioactive peptides. The identified FQHPSFI peptide may be used for the modulation of LPS-stimulated MIP-2 production in AMs....
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PMID: 20186460
PDF is available here.
PDF is available here.
Molecular Immunology (47)11-12 2010
Abstract
We compared AMs from diabetic and non-diabetic rats for phagocytosis via Fc gamma R and the roles of LTs and insulin. Diabetes was induced in male Wistar rats by alloxan (42 mg/kg, i.v.); macrophages were obtained by bronchoalveolar lavage and IgG-opsonised sheep red blood cells (IgG-SRBC) were used...
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PMID: 20510456
PDF is available here.
PDF is available here.
Virus Research (151)1 2010
Abstract
We first established sublines of PAM cells to stably express the PRRSV N protein and assessed alterations in cellular protein productions of N-expressing PAM (PAM-pCD163-N) cells at different time courses by the use of proteomic analysis. A total of 23 protein spots were initially found to be differ...
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PMID: 20394785
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.
FASEB Journal (24)7 2010
Abstract
We studied the effects of elevated P(CO2) on macrophage innate immune responses. In differentiated human THP-1 macrophages and human and mouse alveolar macrophages stimulated with lipopolysaccharide (LPS) and other Toll-like receptor ligands, hypercapnia inhibited expression of tumor necrosis factor...
|
PMID: 20181940
PDF is available here.
PDF is available here.