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Welcome to the Pubget Medical Subject Headings (MESH) browser. Click on a topic or subtopic below to explore related papers. In the gold box is the most recent paper about the current MESH term, and below are 20 related papers.
Gene Expression Regulation (18):
- Up-Regulation
- Gene Expression Regulation, Viral
- Gene Expression Regulation, Enzymologic
- Epistasis, Genetic
- Gene Expression Regulation, Plant
- Gene Expression Regulation, Fungal
- Gene Expression Regulation, Bacterial
- Gene Expression Regulation, Neoplastic
- Gene Amplification
- Down-Regulation
- Transcriptional Activation
- RNA Processing, Post-Transcriptional
- Protein Modification, Translational
- Gene Expression Regulation, Archaeal
- Gene Expression Regulation, Developmental
- Chromatin Assembly and Disassembly
- Epigenesis, Genetic
- Frameshifting, Ribosomal
Recent article
Gene Expression Regulation
Nature Medicine (17)12 2011
Abstract
We identify a central role for the inflammatory complement system in the pathogenesis of osteoarthritis. Through proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with osteoarthritis, we find that expression and activation of complement is abnormally high in hum...
|
PMID: 22057346
PDF is available here.
PDF is available here.
| < More recent | Older article > |
Nature Medicine (17)12 2011
Abstract
We show that nuclear factor-κB1 (NF-κB1) is crucial for maintaining the resting state of DCs. Self-antigen-pulsed unstimulated DCs that do not express NF-κB1 were able to activate CD8(+) T lymphocytes and induce autoimmunity. We further show that NF-κB1 negatively regulates the spontaneous produ...
|
PMID: 22081022
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We generated cortical neuronal precursor cells and neurons from induced pluripotent stem cells derived from individuals with Timothy syndrome. Cells from these individuals have defects in calcium (Ca(2+)) signaling and activity-dependent gene expression. They also show abnormalities in differentiati...
|
PMID: 22120178
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We identify a central role for the inflammatory complement system in the pathogenesis of osteoarthritis. Through proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with osteoarthritis, we find that expression and activation of complement is abnormally high in hum...
|
PMID: 22057346
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We show that nuclear factor-κB1 (NF-κB1) is crucial for maintaining the resting state of DCs. Self-antigen-pulsed unstimulated DCs that do not express NF-κB1 were able to activate CD8(+) T lymphocytes and induce autoimmunity. We further show that NF-κB1 negatively regulates the spontaneous produ...
|
PMID: 22081022
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We generated cortical neuronal precursor cells and neurons from induced pluripotent stem cells derived from individuals with Timothy syndrome. Cells from these individuals have defects in calcium (Ca(2+)) signaling and activity-dependent gene expression. They also show abnormalities in differentiati...
|
PMID: 22120178
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We identify a central role for the inflammatory complement system in the pathogenesis of osteoarthritis. Through proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with osteoarthritis, we find that expression and activation of complement is abnormally high in hum...
|
PMID: 22057346
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We identify a central role for the inflammatory complement system in the pathogenesis of osteoarthritis. Through proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with osteoarthritis, we find that expression and activation of complement is abnormally high in hum...
|
PMID: 22057346
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We show that nuclear factor-κB1 (NF-κB1) is crucial for maintaining the resting state of DCs. Self-antigen-pulsed unstimulated DCs that do not express NF-κB1 were able to activate CD8(+) T lymphocytes and induce autoimmunity. We further show that NF-κB1 negatively regulates the spontaneous produ...
|
PMID: 22081022
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We generated cortical neuronal precursor cells and neurons from induced pluripotent stem cells derived from individuals with Timothy syndrome. Cells from these individuals have defects in calcium (Ca(2+)) signaling and activity-dependent gene expression. They also show abnormalities in differentiati...
|
PMID: 22120178
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We show that nuclear factor-κB1 (NF-κB1) is crucial for maintaining the resting state of DCs. Self-antigen-pulsed unstimulated DCs that do not express NF-κB1 were able to activate CD8(+) T lymphocytes and induce autoimmunity. We further show that NF-κB1 negatively regulates the spontaneous produ...
|
PMID: 22081022
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We generated cortical neuronal precursor cells and neurons from induced pluripotent stem cells derived from individuals with Timothy syndrome. Cells from these individuals have defects in calcium (Ca(2+)) signaling and activity-dependent gene expression. They also show abnormalities in differentiati...
|
PMID: 22120178
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We identify a central role for the inflammatory complement system in the pathogenesis of osteoarthritis. Through proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with osteoarthritis, we find that expression and activation of complement is abnormally high in hum...
|
PMID: 22057346
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We show that nuclear factor-κB1 (NF-κB1) is crucial for maintaining the resting state of DCs. Self-antigen-pulsed unstimulated DCs that do not express NF-κB1 were able to activate CD8(+) T lymphocytes and induce autoimmunity. We further show that NF-κB1 negatively regulates the spontaneous produ...
|
PMID: 22081022
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We generated cortical neuronal precursor cells and neurons from induced pluripotent stem cells derived from individuals with Timothy syndrome. Cells from these individuals have defects in calcium (Ca(2+)) signaling and activity-dependent gene expression. They also show abnormalities in differentiati...
|
PMID: 22120178
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We identify a central role for the inflammatory complement system in the pathogenesis of osteoarthritis. Through proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with osteoarthritis, we find that expression and activation of complement is abnormally high in hum...
|
PMID: 22057346
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We show that nuclear factor-κB1 (NF-κB1) is crucial for maintaining the resting state of DCs. Self-antigen-pulsed unstimulated DCs that do not express NF-κB1 were able to activate CD8(+) T lymphocytes and induce autoimmunity. We further show that NF-κB1 negatively regulates the spontaneous produ...
|
PMID: 22081022
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We generated cortical neuronal precursor cells and neurons from induced pluripotent stem cells derived from individuals with Timothy syndrome. Cells from these individuals have defects in calcium (Ca(2+)) signaling and activity-dependent gene expression. They also show abnormalities in differentiati...
|
PMID: 22120178
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We identify a central role for the inflammatory complement system in the pathogenesis of osteoarthritis. Through proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with osteoarthritis, we find that expression and activation of complement is abnormally high in hum...
|
PMID: 22057346
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We show that nuclear factor-κB1 (NF-κB1) is crucial for maintaining the resting state of DCs. Self-antigen-pulsed unstimulated DCs that do not express NF-κB1 were able to activate CD8(+) T lymphocytes and induce autoimmunity. We further show that NF-κB1 negatively regulates the spontaneous produ...
|
PMID: 22081022
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We generated cortical neuronal precursor cells and neurons from induced pluripotent stem cells derived from individuals with Timothy syndrome. Cells from these individuals have defects in calcium (Ca(2+)) signaling and activity-dependent gene expression. They also show abnormalities in differentiati...
|
PMID: 22120178
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We identify a central role for the inflammatory complement system in the pathogenesis of osteoarthritis. Through proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with osteoarthritis, we find that expression and activation of complement is abnormally high in hum...
|
PMID: 22057346
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We identify a central role for the inflammatory complement system in the pathogenesis of osteoarthritis. Through proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with osteoarthritis, we find that expression and activation of complement is abnormally high in hum...
|
PMID: 22057346
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We show that nuclear factor-κB1 (NF-κB1) is crucial for maintaining the resting state of DCs. Self-antigen-pulsed unstimulated DCs that do not express NF-κB1 were able to activate CD8(+) T lymphocytes and induce autoimmunity. We further show that NF-κB1 negatively regulates the spontaneous produ...
|
PMID: 22081022
PDF is available here.
PDF is available here.
Nature Medicine (17)12 2011
Abstract
We show that nuclear factor-κB1 (NF-κB1) is crucial for maintaining the resting state of DCs. Self-antigen-pulsed unstimulated DCs that do not express NF-κB1 were able to activate CD8(+) T lymphocytes and induce autoimmunity. We further show that NF-κB1 negatively regulates the spontaneous produ...
|
PMID: 22081022
PDF is available here.
PDF is available here.