Outside-to-inside signaling through transmembrane tumor necrosis factor reverses pathologic interleukin-1beta production and deficient apoptosis of rheumatoid arthritis monocytes.

doi:10.1002/art.24778

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Outside-to-inside signaling through transmembrane tumor necrosis factor reverses pathologic interleukin-1beta production and deficient apoptosis of rheumatoid arthritis monocytes.

Undine U Meusch, Manuela M Rossol, Christoph C Baerwald, Sunna S Hauschildt and Ulf U Wagner Arthritis Rheum 60(9):2612-21 (2009) PMID 19714631

Monocytes are a major source of proinflammatory cytokines in rheumatoid arthritis (RA), and inhibitors of monocytic cytokines are highly efficient agents for treatment of the disease. The aim of this study was to analyze the effects of a therapeutic anti-tumor necrosis factor alpha (anti-TNFalpha) antibody on monocytes from patients with RA and healthy control subjects.

DOI: 10.1002/art.24778
Version: za2963e q8za8 q8zbe q8zcb q8zd6 q8ze0 q8zf4 q8zgd

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Outside-to-inside signaling through transmembrane tumor necrosis factor reverses pathologic interleukin-1beta production and deficient apoptosis of rheumatoid arthritis monocytes.

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