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Agouti Signaling ProteinFollow by RSS 

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keywords > Amino Acids, Peptides, and Proteins > Peptides > Intercellular Signaling Peptides and Proteins > Agouti Signaling Protein

Latest papers

Chemical disulfide mapping identifies an inhibitor cystine knot in the agouti signaling protein.

A -defensin mutation causes black coat color in domestic dogs.

Genetics. Beta-defensin repertoire expands.

Genes affecting coat colour and pattern in domestic dogs: a review.

Controlling elements are wild cards in the epigenomic deck.

[Repeating of emotional stress prevents development of melanocortin obesity and type 2 diabetes in the mice with the Agouti yellow mutation].

Gene expression analysis of melanocortin system in vitiligo

Agouti signaling protein stimulates cell division in "viable yellow" (A(vy)/a) mouse liver.

Diet-induced hypermethylation at agouti viable yellow is not inherited transgenerationally through the female.

Proteochemometric analysis of small cyclic peptides' interaction with wild-type and chimeric melanocortin receptors.

The A y allele at the agouti locus enhances sensitivity to endotoxin-induced lethality in mice.

Effects of weaning and weaning weight on neuroendocrine regulators of feed intake in pigs.

Cocaine- and amphetamine-regulated transcript (CART) is colocalized with the orexigenic neuropeptide Y and agouti-related protein and absent from the anorexigenic alpha-melanocyte-stimulating hormone neurons in the infundibular nucleus of the human hypothalamus.

Obesity potentiates development of fatty liver and insulin resistance, but not atherosclerosis, in high-fat diet-fed agouti LDLR-deficient mice.

Linkage and segregation analysis of black and brindle coat color in domestic dogs.

Genotype versus phenotype: Human pigmentation

Mahogany, blood-brain barrier, and fat mass surge in AVY mice.

Modifiers of epigenetic reprogramming show paternal effects in the mouse.

Plasma agouti-related protein (AGRP), growth hormone, insulin responses to a single circuit-resistance exercise in male college students.

Prenatal influences on susceptibility to diet-induced obesity are mediated by altered neuroendocrine gene expression.

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