Latest papers

Spiny mice (Acomys cahirinus) do not respond to thymus-independent type 2 antigens.

Defective microarchitecture of the spleen marginal zone and impaired response to a thymus-independent type 2 antigen in mice lacking scavenger receptors MARCO and SR-A.

Antibody repertoire development in fetal and neonatal piglets. IX. Three pathogen-associated molecular patterns act synergistically to allow germfree piglets to respond to type 2 thymus-independent and thymus-dependent antigens.

Problems in using statistical analysis of replacement and silent mutations in antibody genes for determining antigen-driven affinity selection.

Single and combined deletions of the NTAL/LAB and LAT adaptors minimally affect B-cell development and function.

[Cellular mechanisms implicated in anti-erythrocyte alloimmunization].

Insulin-like growth factor-1 controls type 2 T cell-independent B cell response.

Cutting edge: antibody production to pneumococcal polysaccharides requires CD1 molecules and CD8+ T cells.

Vaccination against encapsulated bacteria in humans: paradoxes.

Normal immune system development in mice lacking the Deltex-1 RING finger domain.

[Isotype switch in the response against T independent antigens by co-inoculation with T dependent antigens].

The role of B cells and accessory cells in the neonatal response to TI-2 antigens.

Rapid induction of splenic and peritoneal B-1a cells in adult mice by thymus-independent type-2 antigen.

The capsule of Bacillus anthracis behaves as a thymus-independent type 2 antigen.

B1b Lymphocytes Confer T Cell-Independent Long-Lasting Immunity

A new murine model of humoral immuno-deficiency specifically affects class switching to T-independent antigens.

The role of adjuvant on the regulatory effects of NK cells on B cell responses as revealed by a new model of NK cell deficiency.

T cell-dependent and -independent responses.

Increased numbers of B-1 cells and enhanced responses against TI-2 antigen in mice lacking APS, an adaptor molecule containing PH and SH2 domains.

Disparate in vitro and in vivo requirements for IL-2 during antigen-independent CD8 T cell expansion.