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  • Anoikis

ApoptosisFollow by RSS 

You've reached a pubget topic page. Below are the latest papers on this topic, with subtopics on the left.

keywords > Cell Physiological Phenomena > Cell Physiological Processes > Cell Death > Apoptosis

Latest papers

Quality characteristics and fertilizing ability of ram sperm subpopulations separated by partition in an aqueous two-phase system

ER stress and apoptosis: a new mechanism for retinal cell death.

PrP antibodies do not trigger mouse hippocampal neuron apoptosis.

[Biomarker for bladder cancer].

Identification of phosphorylated serine-15 and -82 residues of HSPB1 in 5-fluorouracil-resistant colorectal cancer cells by proteomics.

Nilotinib and MEK Inhibitors Induce Synthetic Lethality through Paradoxical Activation of RAF in Drug-Resistant Chronic Myeloid Leukemia

SAG/RBX2/ROC2 E3 Ubiquitin Ligase Is Essential for Vascular and Neural Development by Targeting NF1 for Degradation.

Cordycepin-induced apoptosis and autophagy in breast cancer cells are independent of the estrogen receptor.

Interferon-α2b and transforming growth factor-β1 treatments on HCC cell lines: Are Wnt/β-catenin pathway and Smads signaling connected in hepatocellular carcinoma?

Bilateral inhibition of HAUSP deubiquitinase by a viral interferon regulatory factor protein.

BAX unleashed: the biochemical transformation of an inactive cytosolic monomer into a toxic mitochondrial pore.

Inhibition of proteasome deubiquitinating activity as a new cancer therapy.

Heme oxygenase-1 induction protects the heart and modulates cellular and extracellular remodelling after myocardial infarction in rats.

Human bone marrow-derived stem cell proliferation is inhibited by hepatocyte growth factor via increasing the cell cycle inhibitors p53, p21 and p27.

Apoptotic ciliated cells on sputum smear.

Dihydrofolate reductase is required for the development of heart and outflow tract in zebrafish.

Exploiting oncogene-induced replicative stress for the selective killing of Myc-driven tumors.

Focal adhesion kinase inhibitors are potent anti-angiogenic agents.

Biological leukemia treatment shows breakthrough potential.

Intracellular ATP levels determine cell death fate of cancer cells exposed to both standard and redox chemotherapeutic agents.

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