Latest papers

The role of the pro-apoptotic protein Siva in the pathogenesis of Familial Mediterranean fever: A structural and functional analysis.

HMGB1: a novel Beclin 1-binding protein active in autophagy.

siRNA nanoparticle functionalization of nanostructured scaffolds enables controlled multilineage differentiation of stem cells.

Astaxanthin upregulates heme oxygenase-1 expression through ERK1/2 pathway and its protective effect against beta-amyloid-induced cytotoxicity in SH-SY5Y cells

Overexpression of Bax inhibitor-1 (BI-1) induces cell transformation in NIH3T3 cells.

Excessive apoptosis as a downstream molecular event during the development of congenital heart diseases.

Epigenetic silencing of BIM in glucocorticoid poor-responsive pediatric acute lymphoblastic leukemia, and its reversal by histone deacetylase inhibition.

NF-kappaB inhibits T-cell activation-induced, p73-dependent cell death by induction of MDM2.

Induction of cellular apoptosis in human breast cancer by DLBS1425, a Phaleria macrocarpa compound extract, via down-regulation of PI3-kinase/AKT pathway.

Beclin 1 over- and underexpression in colorectal cancer: distinct patterns relate to prognosis and tumour hypoxia.

Program death-1 signaling and regulatory T cells collaborate to resist the function of adoptively transferred cytotoxic T lymphocytes in advanced acute myeloid leukemia.

Reversion of epigenetically mediated BIM silencing overcomes chemoresistance in Burkitt lymphoma.

Autophagic degradation of active caspase-8: a crosstalk mechanism between autophagy and apoptosis.

Prevention of cytokine withdrawal-induced apoptosis by Mcl-1 requires interaction between Mcl-1 and Bim.

Effects of ILTG on DAPK1 promoter methylation in colon and leukemia cancer cell lines.

Increase of programmed death-1-expressing intratumoral CD8 T cells predicts a poor prognosis for nasopharyngeal carcinoma.

Decreased level of PDCD4 (programmed cell death 4) protein activated cell proliferation in the lung of A/J mouse.

Grb10 interacts with Bim L and inhibits apoptosis.

B and T lymphocyte attenuator is highly expressed on CMV-specific T cells during infection and regulates their function.

Disparate chromatin landscapes and kinetics of inactivation impact differential regulation of p53 target genes.