Latest papers

Characterization of chemoreceptive protein binding to an oviposition stimulant using a fluorescent micro-binding assay in a butterfly

Increased mortality risk for cancers of the kidney and other urinary organs among Chinese herbalists.

Improved preparation and identification of aristolochic acid-DNA adducts by solid-phase extraction with liquid chromatography-tandem mass spectrometry.

Aristolochic acid nephropathy due to herbal drug intake manifested differently as Fanconi's syndrome and end-stage renal failure--a 7-year follow-up.

Gene expression profiles modulated by the human carcinogen aristolochic acid I in human cancer cells and their dependence on TP53

[Effects of exogenous epidermal growth factor (EGF) on growth inhibition induced by aristolochic acid I (AA-I) in renal proximal tubular epithelial cells].

Human cytochromes P450 1A1 and 1A2 participate in detoxication of carcinogenic aristolochic acid.

[Study of pharmacokinetics of aristolochic acid I and II in rats].

Induction of P450 1A by 3-methylcholanthrene protects mice from aristolochic acid-I-induced acute renal injury.

Comparative study on the aristolochic acid I content of Herba Asarifor safe use.

Final report on carcinogens background document for aristolochic acids.

[Effects of cytochrome P450 isozymes on aristolochic acid renal cytotoxicity].

Species-specific toxicity of aristolochic acid (AA) in vitro.

Patterns of interstitial inflammation during the evolution of renal injury in experimental aristolochic acid nephropathy.

Involvement of p38 MAPK phosphorylation and nitrate formation in aristolochic acid-mediated antiplatelet activity.

The phospholipase A inhibitor, aristolochic acid, disrupts cortical microtubule arrays and root growth in Arabidopsis.

Protective effect of erythropoietin against aristolochic acid-induced apoptosis in renal tubular epithelial cells.

Aristolochic acid nephropathy: a worldwide problem.

Antagonistic, stage-specific selection on defensive chemical sequestration in a toxic butterfly.

Ischemic injury underlies the pathogenesis of aristolochic acid-induced acute kidney injury.