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AtherosclerosisFollow by RSS 

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keywords > Cardiovascular Diseases > Vascular Diseases > Arterial Occlusive Diseases > Arteriosclerosis > Atherosclerosis

Latest papers

Chronic granulomatous disease.

Change of diet, plasma lipids, lipoproteins, and fatty acids during ramadan: a controversial association of the considered Ramadan model with atherosclerosis risk.

Orally administered eicosapentaenoic acid induces rapid regression of atherosclerosis via modulating the phenotype of dendritic cells in LDL receptor-deficient mice.

High platelet count as a link between renal cachexia and cardiovascular mortality in end-stage renal disease patients.

Decreased gene expression of LC3 in peripheral leucocytes of patients with coronary artery disease.

Eicosapentaenoic acid and regression of atherosclerotic lesions: a role for dendritic cells.

Hypertension and CKD.

Macrophage Elovl6 deficiency ameliorates foam cell formation and reduces atherosclerosis in low-density lipoprotein receptor-deficient mice.

Genetics in arterial calcification: pieces of a puzzle and cogs in a wheel.

Microparticles, vascular function, and atherothrombosis.

PARP inhibition in atherosclerosis and its effects on dendritic cells, T cells and auto-antibody levels.

Macrovascular effects and safety issues of therapies for type 2 diabetes.

Transcriptional fingerprint of human whole blood at the site of coronary occlusion in acute myocardial infarction.

Gene-activation mechanisms in the regression of atherosclerosis, elimination of diabetes type 2, and prevention of dementia.

Salutary effects of hemodialysis on low-density lipoprotein proinflammatory and high-density lipoprotein anti-inflammatory properties in patient with end-stage renal disease.

Complement proteins C3 and C4 bind to collagen and elastin in the vascular wall: a potential role in vascular stiffness and atherosclerosis.

In vivo oxidation, platelet activation and simultaneous occurrence of natural immunity in atherosclerosis-prone mice.

Platelets as initiators and mediators of inflammation at the vessel wall.

The hemostatic system as a modulator of atherosclerosis.

Macrophages in the pathogenesis of atherosclerosis.

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