Latest papers

Application of the improved BALB/c 3T3 cell transformation assay to the examination of the initiating and promoting activities of chemicals: the second interlaboratory collaborative study by the non-genotoxic carcinogen study group of Japan.

Comparison of the tumorigenic potential of liver and kidney tumors induced by N-nitrosodimethylamine.

Evidence of oxidative stress and associated DNA damage, increased proliferative drive, and altered gene expression in rat liver produced by the cholangiocarcinogenic agent furan.

Revisiting alternative approaches to the assessment of carcinogenic risk.

Amended Safety Assessment of Sodium Picramate and Picramic Acid

Final Report on the Safety Assessment of Amino Nitrophenols as Used in Hair Dyes

Commentary on Evaluation of possible carcinogenic risk to humans based on liver tumors in rodent assays: the two-year bioassay is no longer necessary.

The mouse carcinogenicity study is no longer a scientifically justifiable core data requirement for the safety assessment of pesticides.

Cellular proliferation, cell death, and liver histology in Gambusia affinis after dietary exposure to benzidine and 2-aminofluorene.

Some aromatic amines, organic dyes, and related exposures.

Genotoxicity and mutagenicity of Rosmarinus officinalis (Labiatae) essential oil in mammalian cells in vivo.

Carbon black, titanium dioxide, and talc.

The study of the tumor stem cell properties of CD133+CD44+ cells in the human lung adenocarcinoma cell line A549.

Murine oncogenicity and pharmacokinetics studies of 9-cis-UAB30, an RXR agonist, for breast cancer chemoprevention.

Evaluation of possible carcinogenic risk to humans based on liver tumors in rodent assays: the two-year bioassay is no longer necessary.

Comparison of data used for setting occupational exposure limits.

Carcinogenesis, bioassays, and the regulatory modus operandi.

Inadequate toxicity tests of food additive acesulfame.

[Efficiency of evaluating the carcinogenicity of chemical substances in short-term tests and the SAR model].

Mechanisms of nuclear vitamin D receptor resistance in Harvey-ras-transfected cells.