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Cell HypoxiaFollow by RSS 

You've reached a pubget topic page. Below are the latest papers on this topic, with subtopics on the left.

keywords > Metabolic Phenomena > Metabolism > Cell Respiration > Cell Hypoxia

Latest papers

HIF-1α mediates tumor hypoxia to confer a perpetual mesenchymal phenotype for malignant progression.

Celecoxib can induce vascular endothelial growth factor expression and tumor angiogenesis.

Pathogenesis of tubular interstitial nephritis.

Attenuated expression and function of the RECK tumor suppressor under hypoxic conditions is mediated by the MAPK signaling pathways.

Hypoxic remodelling of Ca2+ stores does not alter human cardiac myofibroblast invasion.

Loss of ATM/Chk2/p53 pathway components accelerates tumor development and contributes to radiation resistance in gliomas.

Hypoxia increases Annexin A2 expression in osteoblastic cells via VEGF and ERK.

Hypoxia induced upregulation and function of the thiamine transporter, SLC19A3 in a breast cancer cell line.

Rakicidin A effectively induces apoptosis in hypoxia adapted Bcr-Abl positive leukemic cells.

Ischemia-induced angiogenesis is impaired in aminopeptidase A deficient mice via down-regulation of HIF-1α.

Excessive Na+/H+ exchange in disruption of dendritic Na+ and Ca2+ homeostasis and mitochondrial dysfunction following in vitro ischemia.

The basic science of vascular biology: implications for the practicing surgeon.

Computer simulation of tumour control probabilities after irradiation for varying intrinsic radio-sensitivity using a single cell based model.

Controlling matrix formation and cross-linking by hypoxia in cardiovascular tissue engineering.

TCP modelling - why is it important?

[Tumor angiogenesis: recent progress and remaining challenges].

ABCG2 transports and transfers heme to albumin through its large extracellular loop.

The protective effect of 17β-estradiol postconditioning against hypoxia/reoxygenation injury in human gastric epithelial cells

PIASy stimulates HIF1α SUMOylation and negatively regulates HIF1α activity in response to hypoxia.

Neuron specific metabolic adaptations following multi-day exposures to oxygen glucose deprivation.

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