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Contact InhibitionFollow by RSS 

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keywords > Cell Physiological Phenomena > Cell Physiological Processes > Contact Inhibition

Latest papers

Tetraspanin TM4SF5 mediates loss of contact inhibition through epithelial-mesenchymal transition in human hepatocarcinoma.

Endothelial adherens and tight junctions in vascular homeostasis, inflammation and angiogenesis.

Cell contact-inhibition signaling as part of wound-healing processes in brain.

The evolution of contact-dependent inhibition in non-growing populations of Escherichia coli.

Attenuation of p38 MAPK activity upon contact inhibition in fibroblasts.

Inactivation of YAP oncoprotein by the Hippo pathway is involved in cell contact inhibition and tissue growth control.

SND1, a component of RNA-induced silencing complex, is up-regulated in human colon cancers and implicated in early stage colon carcinogenesis.

Insulin-like growth factor II receptor-mediated intracellular retention of cathepsin B is essential for transformation of endothelial cells by Kaposi's sarcoma-associated herpesvirus.

Contact inhibition of migrating lens epithelial cells at the capsular bend created by a sharp-edged intraocular lens after cataract surgery.

Neuropilin-1 promotes unlimited growth of ovarian cancer by evading contact inhibition.

Upregulation of PHLDA2 in Dicer knockdown HEK293 cells.

A simple cell motility assay demonstrates differential motility of human periodontal ligament fibroblasts, gingival fibroblasts, and pre-osteoblasts.

Cell fate determination factor DACH1 inhibits c-Jun-induced contact-independent growth.

Constitutive CD40 signaling phenocopies the transforming function of the Epstein-Barr virus oncoprotein LMP1 in vitro.

Enhancement of cell proliferation in various mammalian cell lines by gene insertion of a cyclin-dependent kinase homolog.

Characterization of telomerase-immortalized, non-neoplastic, human Barrett's cell line (BAR-T).

Analysis of transformation and tumorigenicity using mouse embryonic fibroblast cells.

Cytotoxic G-rich oligodeoxynucleotides: putative protein targets and required sequence motif.

CD4+ CD25high regulatory T cells reduce T cell transendothelial migration in cancer patients.

SDF-1alpha regulation in breast cancer cells contacting bone marrow stroma is critical for normal hematopoiesis.

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