Latest papers

Imbalance between pSmad3 and Notch induces CDK inhibitors in old muscle stem cells.

Identifying the differential effects of silymarin constituents on cell growth and cell cycle regulatory molecules in human prostate cancer cells.

Targeted degradation of the cyclin-dependent kinase inhibitor ICK4/KRP6 by RING-type E3 ligases is essential for mitotic cell cycle progression during Arabidopsis gametogenesis.

G1 arrest and expression of cyclin-dependent kinase inhibitors in tamoxifen-treated MCF-7 human breast cancer cells.

Progression elevated gene-3 promoter (PEG-Prom) confers cancer cell selectivity to human polynucleotide phosphorylase (hPNPase(old-35))-mediated growth suppression.

The clinical application of autologous bioengineered skin based on a hyaluronic acid scaffold.

Phosphorylation of nm23-H1 by CKI induces its complex formation with h-prune and promotes cell motility.

Degradation of the cyclin-dependent kinase inhibitor KRP1 is regulated by two different ubiquitin E3 ligases.

The effects of cyclin-dependent kinase inhibitors on adipogenic differentiation of human mesenchymal stem cells.

Reactivation of methylation-silenced tumor suppressor gene p16INK4a by nordihydroguaiaretic acid and its implication in G1 cell cycle arrest.

CDK inhibitors: cell cycle regulators and beyond.

Transcriptional control of cell-cycle quiescence during C. elegans development

Distinct HDACs regulate the transcriptional response of human cyclin-dependent kinase inhibitor genes to Trichostatin A and 1alpha,25-dihydroxyvitamin D3.

Progesterone receptor isoform functions in normal breast development and breast cancer.

Inhibition of the Ras oncoprotein reduces proliferation of hepatocytes in vitro and in vivo in rats.

Aberrant methylation in promoter regions of cyclin-dependent kinase inhibitor genes in adenoid cystic carcinoma of the salivary gland.

The G2/M transition in eukaryotes.

The CDK inhibitors: potential targets for therapeutic stem cell manipulations?

Simvastatin inhibits cell cycle progression in glucose-stimulated proliferation of aortic vascular smooth muscle cells by up-regulating cyclin dependent kinase inhibitors and p53.

Thrombopoietin/MPL signaling regulates hematopoietic stem cell quiescence and interaction with the osteoblastic niche.