Latest papers

Disruption of the U(L)41 gene in the herpes simplex virus 2 dl5-29 mutant increases its immunogenicity and protective capacity in a murine model of genital herpes.

[Construction and characterization of hSef recombinant adenoviral vectors]

Third-generation flavivirus vaccines based on single-cycle, encapsidation-defective viruses.

Viral complementation allows HIV-1 replication without integration.

The delayed early gene G23 of temperate mycobacteriophage L1 regulates the expression of deoxyribonuclease, the product of another delayed early gene of the phage.

Use of a defective RNA of broad bean mottle bromovirus for stable gene expression in legumes.

Combining gene and immunotherapy for prostate cancer.

[High-titer preparation of HIV-1-based defective lentivector and it mediated efficient gene transfer].

Immunization with a replication-defective herpes simplex virus 2 mutant reduces herpes simplex virus 1 infection and prevents ocular disease

Determination of virus burst size in vivo using a single-cycle SIV in rhesus macaques.

Using vectors derived from tomato bushy stunt virus (TBSV) and TBSV defective interfering RNAs (DIs).

Antiplatelet drug therapy moderates immune-mediated liver disease and inhibits viral clearance in mice infected with a replication-deficient adenovirus.

Lentiviral vectors: are they the future of animal transgenesis?

Tobacco mosaic virus defective RNAs expressing C-terminal methyltransferase domain sequences are severely impaired in long-distance movement in Nicotiana benthamiana.

Superinfection exclusion in BHK-21 cells persistently infected with Junín virus.

High efficient generation of replication-defective adenoviruses containing thymidine kinase by homogeneous recombination in bacteria.

Successful immunization with a single injection of non-integrating lentiviral vector.

Antitumor effects of non-replicative herpes simplex vectors expressing antiangiogenic proteins and thymidine kinase on Lewis lung carcinoma establishment and growth.

Reciprocal sequence exchange between non-retro viruses and hosts leading to the appearance of new host phenotypes

The substitution U475 --> C with Sabin3-like mutation within the IRES attenuate Coxsackievirus B3 cardiovirulence.