Latest papers

X-chromosome upregulation and inactivation: two sides of the dosage compensation mechanism in mammals.

Highlights from this Issue.

Molecularly severe roX1 mutations contribute to dosage compensation in Drosophila.

Interaction study of the male specific lethal (MSL) complex and trans-acting dosage effects in metafemales of Drosophila melanogaster.

The chromosomal high-affinity binding sites for the Drosophila dosage compensation complex.

Female monozygotic twins discordant for hemophilia A due to nonrandom X-chromosome inactivation.

Sexual antagonism and the evolution of X chromosome inactivation.

Cellular reactions to gene dosage imbalance: genomic, transcriptomic and proteomic effects.

Dosage compensation of the mammalian X chromosome influences the phenotypic variability of X-linked dominant male-lethal disorders.

Kabuki syndrome in a girl with mosaic 45,X/47,XXX and aortic coarctation.

Genome-wide analysis reveals MOF as a key regulator of dosage compensation and gene expression in Drosophila.

New twists in X-chromosome inactivation.

Chromatin Proteins Do Double Duty

Transcription-coupled methylation of histone H3 at lysine 36 regulates dosage compensation by enhancing recruitment of the MSL complex in Drosophila melanogaster.

SU(VAR)3-7 links heterochromatin and dosage compensation in Drosophila.

Biological models for phytochemical research: from cell to human organism.

Comparison of X-chromosome inactivation patterns in multiple tissues from human females.

Fine mapping of regulatory loci for mammalian gene expression using radiation hybrids.

MLE activates transcription via the minimal transactivation domain in Drosophila.

Aberrant DNA methylation associated with bipolar disorder identified from discordant monozygotic twins.