Latest papers

Immunization with a single extracellular enveloped virus protein produced in bacteria provides partial protection from a lethal orthopoxvirus infection in a natural host.

Involvement of Fas and FasL in Ectromelia virus-induced apoptosis in mouse brain.

Classic paper: Fenner on the exanthemata.

Antibodies and CD8+ T cells are complementary and essential for natural resistance to a highly lethal cytopathic virus.

The nonreplicating smallpox candidate vaccines defective vaccinia Lister (dVV-L) and modified vaccinia Ankara (MVA) elicit robust long-term protection.

An orally bioavailable antipoxvirus compound (ST-246) inhibits extracellular virus formation and protects mice from lethal orthopoxvirus Challenge.

Ectromelia virus: the causative agent of mousepox.

Functional analysis of granzyme M and its role in immunity to infection.

Mousepox conjunctivitis: the role of Fas/FasL-mediated apoptosis of epithelial cells in virus dissemination.

The efficacy of cidofovir treatment of mice infected with ectromelia (mousepox) virus encoding interleukin-4.

Biosynthesis of the IFN-gamma binding protein of ectromelia virus, the causative agent of mousepox.

The multidrug resistance gene mdr1a influences resistance to ectromelia virus infection by mechanisms other than conventional immunity.

Polarized type 1 cytokine response and cell-mediated immunity determine genetic resistance to mousepox.

Protective effect of exogenous recombinant mouse interferon-gamma and tumour necrosis factor-alpha on ectromelia virus infection in susceptible BALB/c mice.

Efficacy of oral active ether lipid analogs of cidofovir in a lethal mousepox model.

The genomic sequence of ectromelia virus, the causative agent of mousepox.

Apoptosis during ectromelia orthopoxvirus infection is DEVDase dependent: in vitro and in vivo studies.

Possible mechanism for the enhanced lethality of an interleukin-4-expressing mousepox virus.

Expression of mouse interleukin-4 by a recombinant ectromelia virus suppresses cytolytic lymphocyte responses and overcomes genetic resistance to mousepox.

Virulence of mousepox virus is independent of serpin-mediated control of cellular cytotoxicity.