Latest papers

Heterologous replacement of the supposed host determining region of avihepadnaviruses: high in vivo infectivity despite low infectivity for hepatocytes.

Identification and molecular analysis of the highly pathogenic duck hepatitis virus type 1 in Hubei province of China

Surviving mousepox infection requires the complement system.

Inhibition of duck hepatitis B virus infection of liver cells by combined treatment with viral e antigen and carbohydrates.

Mouse hepatitis virus liver pathology is dependent on ADP-ribose-1''-phosphatase, a viral function conserved in the alpha-like supergroup.

Murine coronavirus mouse hepatitis virus is recognized by MDA5 and induces type I interferon in brain macrophages/microglia.

Genetic diversity of the VP1 gene of duck hepatitis virus type I (DHV-I) isolates from southeast China is related to isolate attenuation.

CD11c identifies a subset of murine liver natural killer cells that responds to adenoviral hepatitis.

Establishment of an infectious genotype 1b hepatitis C virus clone in human hepatocyte chimeric mice.

[The functions of liver natural killer cells in murine fulminant hepatitis induced by murine hepatitis virus strain 3].

Classification of duck hepatitis virus into three genotypes based on molecular evolutionary analysis.

In vitro and in vivo anti-hepatitis B virus activities of a plant extract from Geranium carolinianum L.

Adenoviral hepatitis in a SIV-infected rhesus monkey (Macaca mulatta).

Aggravation of viral hepatitis by platelet-derived serotonin.

Restricted quasispecies variation following infection with the GB virus B.

In vivo and in vitro anti-hepatitis B virus activity of total phenolics from Oenanthe javanica.

Serologic survey of woodchuck hepatitis virus in North Carolina woodchucks (Marmota monax).

DNA vaccination in combination or not with lamivudine treatment breaks humoral immune tolerance and enhances cccDNA clearance in the duck model of chronic hepatitis B virus infection.

Intrahepatic endothelial and Kupffer cells involved in immunosuppressive cytokines and natural killer (NK)/NK T cell disorders in viral acute hepatitis.

Antiviral therapy with entecavir combined with post-exposure "prime-boost" vaccination eliminates duck hepatitis B virus-infected hepatocytes and prevents the development of persistent infection.