Latest papers

mGRASP enables mapping mammalian synaptic connectivity with light microscopy.

Emetine treatment masks initial LTP without affecting long-term stability.

Metabolic gene expression changes in the hippocampus of obese epileptic male rats in the pilocarpine model of temporal lobe epilepsy.

Lithium, phenserine, memantine and pioglitazone reverse memory deficit and restore phospho-GSK3β decreased in hippocampus in intracerebroventricular streptozotocin induced memory deficit model.

Characterization of nectin processing mediated by presenilin-dependent γ-secretase.

Cortistatin is induced in brain tissue and exerts neuroprotection in a rat model of bacterial meningoencephalitis.

Disrupting effect of drug-induced reward on spatial but not cue-guided learning: implication of the striatal protein kinase A/cAMP response element-binding protein pathway.

Use dependence of presynaptic tenacity.

CRP1, a protein localized in filopodia of growth cones, is involved in dendritic growth.

Neuronal P2X2 receptors are mobile ATP sensors that explore the plasma membrane when activated.

[PolyI:C-induced neurodevelopmental animal model for schizophrenia].

Cyclin E Constrains Cdk5 Activity to Regulate Synaptic Plasticity and Memory Formation

Long term potentiation is impaired in membrane glycoprotein CD200-deficient mice: a role for Toll-like receptor activation.

A preliminary study of sex differences in brain activation during a spatial navigation task in healthy adults.

Specific regulatory motifs predict glucocorticoid responsiveness of hippocampal gene expression.

Nitration of tyrosine 10 critically enhances amyloid β aggregation and plaque formation.

Erythropoietin decreases the excitatory neurotransmitter release probability and enhances synaptic plasticity in mice hippocampal slices.

Changes of paired-pulse evoked responses during the development of epileptic activity in the hippocampus.

APP is phosphorylated by TrkA and regulates NGF/TrkA signaling.

Little exercise, big effects: reversing aging and infection-induced memory deficits, and underlying processes.