Latest papers

Atypical Melanocytic Proliferations and New Primary Melanomas in Patients With Advanced Melanoma Undergoing Selective BRAF Inhibition.

Neuropeptide Y expression in cutaneous melanoma

Multicenter Case-Control Study of Risk Factors for Cutaneous Melanoma in Valencia, Spain.

Diagnosis of blue nevus-like metastatic uveal melanoma confirmed by fluorescence in situ hybridization (FISH) for monosomy 3.

A Highly Specific and Discriminatory FISH Assay for Distinguishing Between Benign and Malignant Melanocytic Neoplasms.

Randomized, double-blind, placebo-controlled trial of sulindac in individuals at risk for melanoma: Evaluation of potential chemopreventive activity.

Increased HOX C13 expression in metastatic melanoma progression.

Diverse cutaneous side effects associated with BRAF inhibitor therapy: A clinicopathologic study.

Germline melanoma susceptibility and prognostic genes: A review of the literature.

Effectiveness of combined pulsed dye and Qs-ruby laser treatment for large to giant congenital melanocytic naevi.

A circum-corneal conjunctival nevus in a child.

Availability of digital dermoscopy in daily practice dramatically reduces the number of excised melanocytic lesions: results from an observational study.

Consumption of the epidermis in acral lentiginous melanoma.

Fluorescence In Situ Hybridization (FISH) Analysis of Melanocytic Nevi and Melanomas: Sensitivity, Specificity, and Lack of Association With Sentinel Node Status.

Abrogation of BRAFV600E-induced senescence by PI3K pathway activation contributes to melanomagenesis.

Profiling of ABC transporters ABCB5, ABCF2 and nestin-positive stem cells in nevi, in situ and invasive melanoma.

Accuracy in skin cancer diagnosis: A retrospective study of an Australian public hospital dermatology department.

Micro-Raman Spectroscopy Study of Human Pterygium and Conjunctival Melanocytic Nevi.

Histology-Specific MicroRNA Alterations in Melanoma.

Genetic variants in DNA repair genes and the risk of cutaneous malignant melanoma in melanoma-prone families with/without CDKN2A mutations.