Latest papers

Molecular approaches for eco-epidemiological studies of Paracoccidioides brasiliensis.

Morphologic organization of pulmonary granulomas in mice infected with Paracoccidioides brasiliensis.

Inteins in pathogenic fungi: a phylogenetic tool and perspectives for therapeutic applications.

Preferential transcription of Paracoccidioides brasiliensis genes: host niche and time-dependent expression

Paracoccidioides brasiliensis vaccine formulations based on the gp43-derived P10 sequence and the Salmonella enterica FliC flagellin.

Characterization and functional analysis of the beta-1,3-glucanosyltransferase 3 of the human pathogenic fungus Paracoccidioides brasiliensis.

Inhibition of human neutrophil apoptosis by Paracoccidioides brasiliensis: role of interleukin-8.

Fungal foes: Lacazia loboi.

The malate synthase of Paracoccidioides brasiliensis is a linked surface protein that behaves as an anchorless adhesin.

Identification of Paracoccidioides brasiliensis in adrenal glands biopsies of two patients with paracoccidioidomycosis and adrenal insufficiency.

MyD88 is dispensable for resistance to Paracoccidioides brasiliensis in a murine model of blood-borne disseminated infection.

Deficiency of IL-12p40 subunit determines severe paracoccidioidomycosis in mice.

Protective immunity induced by rPb27 of Paracoccidioides brasiliensis.

Additive effect of P10 immunization and chemotherapy in anergic mice challenged intratracheally with virulent yeasts of Paracoccidioides brasiliensis.

Evidence for the role of calcineurin in morphogenesis and calcium homeostasis during mycelium-to-yeast dimorphism of Paracoccidioides brasiliensis.

Vaccination with heat shock protein 60 induces a protective immune response against experimental Paracoccidioides brasiliensis pulmonary infection.

Phylogenetic analysis of PRP8 intein in Paracoccidioides brasiliensis species complex.

Pseudotumoral neuroparacoccidioidomycosis: one case report.

Role of interleukin (IL)-18 in experimental paracoccidioidomycosis.

Helminth infections associated with multiple sclerosis induce regulatory B cells.