Latest papers

Regression of superficial glomerular podocyte injury in type 2 diabetic rats with overt albuminuria: effect of angiotensin II blockade.

CIN85/RukL is a novel binding partner of nephrin and podocin and mediates slit diaphragm turnover in podocytes.

Activation of the aldosterone/mineralocorticoid receptor system in chronic kidney disease and metabolic syndrome.

Maximal 'CD80-uria' with minimal change.

Morphometric analysis of podocyte foot process effacement in IgA nephropathy and its association with proteinuria.

Actin-depolymerizing factor cofilin-1 is necessary in maintaining mature podocyte architecture.

Deletion of Von Hippel-Lindau in Glomerular Podocytes Results in Glomerular Basement Membrane Thickening, Ectopic Subepithelial Deposition of Collagen α1α2α1(IV), Expression of Neuroglobin, and Proteinuria

Podocyte-specific overexpression of GLUT1 surprisingly reduces mesangial matrix expansion in diabetic nephropathy in mice.

Transgenic overexpression of GLUT1 in mouse glomeruli produces renal disease resembling diabetic glomerulosclerosis.

Tamoxifen-inducible podocyte-specific iCre recombinase transgenic mouse provides a simple approach for modulation of podocytes in vivo.

Plasma cell dyscrasia causing light chain tubulopathy without Fanconi syndrome.

[Bone mesenchymal stem cell transplantation repairs glomerular podocytes in rats with puromycin aminonucleoside-induced nephrosis].

[Novel insights into the glomerular structure]

Molecular make-up of the glomerular filtration barrier

Cilnidipine suppresses podocyte injury and proteinuria in metabolic syndrome rats: possible involvement of N-type calcium channel in podocyte.

Aliskiren inhibits intracellular angiotensin II levels without affecting (pro)renin receptor signals in human podocytes.

Membranous nephropathy: recent travels and new roads ahead.

Mice with podocyte-specific overexpression of wild type alpha-actinin-4 are healthy controls for K256E-alpha-actinin-4 mutant transgenic mice.

A novel WT1 gene mutation in a three-generation family with progressive isolated focal segmental glomerulosclerosis.

Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice.