Latest papers

Infection of lung epithelial cells and induction of pulmonary adenocarcinoma is not the most common outcome of naturally occurring JSRV infection during the commercial lifespan of sheep

An influx of macrophages is the predominant local immune response in ovine pulmonary adenocarcinoma.

Successful induction of ovine pulmonary adenocarcinoma in lambs of different ages and detection of viraemia during the preclinical period.

Coexistence of enzootic nasal adenocarcinoma and jaagsiekte retrovirus infection in sheep.

Detection and characterization of betaretroviral sequences, related to sheep Jaagsiekte virus, in Africans from Nigeria and Cameroon.

Analysis of integration sites of Jaagsiekte sheep retrovirus in ovine pulmonary adenocarcinoma.

Retrovirus-induced lung cancer: mechanisms of transformation of alveolar type II epithelial cells.

Ovine pulmonary adenomatosis in Patagonia, Argentina.

Relevance of Akt phosphorylation in cell transformation induced by Jaagsiekte sheep retrovirus.

Goodbye, Dolly; first cloned sheep dies at six years old.

[The sheep, model for human lung pathology].

Natural history of JSRV in sheep.

Molecular biology of jaagsiekte sheep retrovirus.

Transformation and oncogenesis by jaagsiekte sheep retrovirus.

Identification of Hyal2 as the cell-surface receptor for jaagsiekte sheep retrovirus and ovine nasal adenocarcinoma virus.

Endogenous retroviruses related to jaagsiekte sheep retrovirus.

A history of ovine pulmonary adenocarcinoma (jaagsiekte) and experiments leading to the deduction of the JSRV nucleotide sequence.

The jaagsiekte sheep retrovirus envelope gene induces transformation of the avian fibroblast cell line DF-1 but does not require a conserved SH2 binding domain.

Systemic immune responses following infection with Jaagsiekte sheep retrovirus and in the terminal stages of ovine pulmonary adenocarcinoma.

Lack of DNA evidence for jaagsiekte sheep retrovirus in human bronchioloalveolar carcinoma.