Latest papers

Regulation of the subcellular distribution of key cellular RNA-processing factors during permissive human cytomegalovirus infection.

An antisense microwalk reveals critical role of an intronic position linked to a unique long-distance interaction in pre-mRNA splicing.

Promotion of exon 6 inclusion in HuD pre-mRNA by Hu protein family members.

A genomic-library based discovery of a novel, possibly synthetic, acid-tolerance mechanism in Clostridium acetobutylicum involving non-coding RNAs and ribosomal RNA processing.

PRP-17 and the pre-mRNA splicing pathway are preferentially required for the proliferation versus meiotic development decision and germline sex determination in Caenorhabditis elegans.

Modulation of aberrant NF1 pre-mRNA splicing by kinetin treatment.

Regulation of alternative splicing by histone modifications.

The snoRNA MBII-52 (SNORD 115) is processed into smaller RNAs and regulates alternative splicing.

A simple artificial microRNA vector based on ath-miR169d precursor from Arabidopsis.

Coupling genetics and post-genomic approaches to decipher the cellular splicing code at a systems-wide level.

Molecular detection of viable bacterial pathogens in water by ratiometric pre-rRNA analysis.

Poly(A) signal-dependent degradation of unprocessed nascent transcripts accompanies poly(A) signal-dependent transcriptional pausing in vitro.

Splicing regulation of the survival motor neuron genes and implications for treatment of spinal muscular atrophy.

[Human ribosomal protein S16 inhibites excision of the first intron from its own].

Nuclear networking fashions pre-messenger RNA and primary microRNA transcripts for function

[Interchromatin granule clusters of mouse preovulatory oocytes, organization, molecular composition and possible functions]

Effects of the promoter open complex formation on gene expression dynamics.

A computational-based update on microRNAs and their targets in barley (Hordeum vulgare L.)

Genome-wide approaches to monitor pre-mRNA splicing.

Identification of a MIP mutation that activates a cryptic acceptor splice site in the 3' untranslated region.