Latest papers

Carbon monoxide-releasing molecules protect against ischemia-reperfusion injury during kidney transplantation.

Carbon monoxide-releasing molecules protect against ischemia-reperfusion injury during kidney transplantation.

Mice deficient in pituitary adenylate cyclase activating polypeptide (PACAP) show increased susceptibility to in vivo renal ischemia/reperfusion injury.

Mice deficient in pituitary adenylate cyclase activating polypeptide (PACAP) show increased susceptibility to in vivo renal ischemia/reperfusion injury.

Mice deficient in pituitary adenylate cyclase activating polypeptide (PACAP) show increased susceptibility to in vivo renal ischemia/reperfusion injury.

Early proteins E6 and E7 of human papillomavirus may attenuate ischemia-reperfusion injury.

Early proteins E6 and E7 of human papillomavirus may attenuate ischemia-reperfusion injury.

Early proteins E6 and E7 of human papillomavirus may attenuate ischemia-reperfusion injury.

Impairment of IGF-I expression and anabolic signaling following ischemia/reperfusion in skeletal muscle of old mice.

Crosstalk between complement and Toll-like receptor activation in relation to donor brain death and renal ischemia-reperfusion injury.

Improving long-term outcome in allograft transplantation: role of ionic composition and polyethylene glycol.

Improving long-term outcome in allograft transplantation: role of ionic composition and polyethylene glycol.

Evaluation of kidney repair capacity using 99mTc-DMSA in ischemia/reperfusion injury models.

Evaluation of kidney repair capacity using 99mTc-DMSA in ischemia/reperfusion injury models.

Evaluation of kidney repair capacity using 99mTc-DMSA in ischemia/reperfusion injury models.

Beneficial effects of hydrogen gas against spinal cord ischemia-reperfusion injury in rabbits.

Effects of intravenous sulfide during porcine aortic occlusion-induced kidney ischemia/reperfusion injury.

Effects of intravenous sulfide during porcine aortic occlusion-induced kidney ischemia/reperfusion injury.

Endoplasmic reticulum stress is a mediator of posttransplant injury in severely steatotic liver allografts.

The antioxidant EPC-K1 attenuates renal ischemia-reperfusion injury in a rat model.