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Retrograde DegenerationFollow by RSS 

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keywords > Pathological Conditions, Signs and Symptoms > Pathologic Processes > Nerve Degeneration > Retrograde Degeneration

Latest papers

Metabotropic suppression of excitation in murine autaptic hippocampal neurons.

[Mechanisms of development of secondary axonal failure in diffuse and local demyelinating neuropathies].

Vasopressors are essential during cardiopulmonary resuscitation in rats: Is vasopressin superior to adrenaline?

Chondroitinase applied to peripheral nerve repair averts retrograde axonal regeneration.

Major myelin protein gene (P0) mutation causes a novel form of axonal degeneration.

Up-regulation of PK11195 binding in areas of axonal degeneration coincides with early microglial activation in mouse brain.

Induction of autophagy in axonal dystrophy and degeneration.

Neurotrophins support regenerative axon assembly over CSPGs by an ECM-integrin-independent mechanism.

Optic nerve atrophy and retinal nerve fibre layer thinning following optic neuritis: evidence that axonal loss is a substrate of MRI-detected atrophy.

Neuronal damage in hydrocephalus and its restoration by shunt insertion in experimental hydrocephalus: a study involving the neurofilament-immunostaining method.

Angiotensin converting enzyme has an inhibitory role in CGRP metabolism in human skin.

Traumatic axonal injury in the perisomatic domain triggers ultrarapid secondary axotomy and Wallerian degeneration.

Retinoic acid receptor-dependent survival of olfactory sensory neurons in postnatal and adult mice.

Retinal ganglion cell activity from the multifocal electroretinogram in pig: optic nerve section, anaesthesia and intravitreal tetrodotoxin.

Effects of axotomy on telomere length, telomerase activity, and protein in activated microglia.

Retrograde degeneration of the corticospinal tract associated with pontine infarction.

Treatment of passive experimental autoimmune encephalomyelitis in SJL mice with a recombinant TCR ligand induces IL-13 and prevents axonal injury.

A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde manner.

Abducens internuclear neurons depend on their target motoneurons for survival during early postnatal development.

A transcriptional role for C/EBP β in the neuronal response to axonal injury

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